Cangrelor
Indications
Cangrelor is used for:
Indicated as an adjunct to percutaneous coronary intervention (PCI) to reduce the risk of periprocedural myocardial infarction (MI), repeat coronary revascularization, and stent thrombosis (ST) in patients who have not been treated with a P2Y12 platelet inhibitor and are not being given a glycoprotein IIb/IIIa inhibitor
Adult Dose
Percutaneous Coronary Intervention
30 mcg/kg IV bolus infused over 1 minute before PCI, THEN
Immediately follow bolus injection with 4 mcg/kg/min IV infusion; continue for at least 2 hr or duration of PCI, whichever is longer
Transition patients to oral P2Y12 platelet inhibitor
Choose from 1 of the loading-dose regimens described below to initiate oral therapy:
Ticagrelor: 180 mg PO at any time during cangrelor infusion or immediately after discontinuation
Prasugrel: 60 mg PO immediately after discontinuing cangrelor; do not administer prasugrel prior to cangrelor discontinuation because of drug interaction
Clopidogrel: 600 mg PO immediately after discontinuing cangrelor; do not administer clopidogrel prior to cangrelor discontinuation because of drug interaction
Child Dose
Renal Dose
Administration
IV Preparation
For each 50 mg/vial, reconstitute by adding 5 mL of sterile water for injection
Swirl gently until all material is dissolved; avoid vigorous mixing
Allow any foam to settle
Ensure that vial contents are fully dissolved and the reconstituted material is a clear, colorless to pale yellow solution
Do not use without dilution
Before administration, each reconstituted vial must be diluted further with 0.9% NaCl or D5W
Withdraw the contents from 1 reconstituted vial and add to 250-mL bag of 0.9% NaCl or D5W
Mix the bag thoroughly
This dilution results in a concentration of 200 mcg/mL and should be sufficient for at least 2 hr of dosing
Patients who weigh ?100 kg require a minimum of 2 bags
IV Administration
Administer via a dedicated IV line
Administer the bolus volume rapidly (<1 minute), from the diluted bag via manual IV push or pump
Ensure the bolus is completely administered before the start of PCI
Start the IV infusion immediately after the bolus administration
Contra Indications
Significant active bleeding
Hypersensitivity
Precautions
Increased risk of bleeding; bleeding events of all severities were more common with cangrelor than with clopidogrel
Lactation: Unknown if distributed in human breast milk; use caution
Pregnancy-Lactation
Pregnancy
There are no available data on use in pregnant women to evaluate for a drug-associated risk of major birth defects, miscarriage or adverse maternal or fetal outcomes; untreated myocardial infarction can be fatal to pregnant women and fetus
Disease-associated maternal and/or embryo/fetal risk
Myocardial infarction is a medical emergency in pregnancy which can be fatal to pregnant woman and fetus if left untreated; life-sustaining therapy for the pregnant woman should not be withheld due to potential concerns regarding the effects of the drug on the fetus
Animal data
In animal reproduction studies, continuous infusion of drug in pregnant rats and rabbits throughout organogenesis at dose approximately 2-times maximum recommended human dose (MRHD) did not result in fetal malformations
Labor or delivery
Drug use during labor and delivery may increase risk for maternal bleeding and hemorrhage; performance of neuraxial blockade procedures is not advised during drug use due to potential risk of spinal hematoma; when possible, discontinue drug 1 hour prior to labor, delivery, or neuraxial blockade
Lactation
There are no data on presence of drug in human milk or animal milk, effects on breastfed infant, or on milk production; due to its short-half life, drug exposure is expected to be very low in breastfed infant
Interactions
Adverse Effects
Side effects of Cangrelor :
Bleeding
Mild (14.9%)
Moderate (0.4%)
Severe/life-threatening (0.2%)
1-10%
Worsening renal function in patients with CrCl <30 mL/min (3.2%)
<1%
Hypersensitivity
Mechanism of Action
P2Y12 platelet inhibitor that blocks ADP-induced platelet activation and aggregation; it binds selectively and reversibly to the P2Y12 receptor to prevent further signaling and platelet activation