Cobimetinib

Indications

Cobimetinib is used for: In combination w/ vemurafenib for the treatment of adult patients w/ unresectable or metastatic melanoma w/ a BRAF V600 mutation.

Adult Dose

Melanoma Indicated for unresectable or metastatic melanoma in patients with a BRAF V600E or V600K mutation, in combination with vemurafenib 60 mg PO qDay for the first 21 days of each 28-day cycle until disease progression or unacceptable toxicity Vemurafenib: 960 mg PO BID on days 1-28 of an every 28-day cycle Hepatic impairment Mild-to-severe (Child-Pugh A to C): No dosage adjustment necessary

Child Dose

Renal Dose

Renal impairment Mild-to-moderate (CrCl 30-89 mL/min): No dosage adjustment necessary Severe (CrCl <30 mL/min): Safety and efficacy not established

Administration

May be taken with or without food.

Contra Indications

Hypersensitivity.

Precautions

Patients must have BRAF V600 mutation +ve tumour status confirmed by a validated test before treatment. Assess symptoms of new or worsening visual disturbances. Evaluate LVEF before initiation, then after the 1st mth of treatment & at least every 3 mth or as clinically indicated until treatment discontinuation. Monitor liver value abnormalities. Diarrhoea, lactose intolerance; QT prolongation. Concomitant use w/ strong/moderate CYP3A inhibitors. Lactation Unknown if distributed in human breast milk Because of the potential for serious adverse reactions in a breastfed infant, advise women not to breastfeed during treatment and for 2 weeks after the final dose

Pregnancy-Lactation

Pregnancy Based on findings from animal reproduction studies and its mechanism of action, cobimetinib can cause fetal harm when administered to a pregnant woman In animal reproduction studies, oral administration of cobimetinib in pregnant rats during organogenesis was teratogenic and embryotoxic at exposures (AUC) that were 0.9- to 1.4-times those observed in humans at the recommended human dose of 60 mg Advise pregnant women of the potential risk to a fetus Contraception: Advise females of reproductive potential to use effective contraception during treatment with and for 2 weeks after the final dose Infertility: Based on findings in animals, cobimetinib may reduce fertility in females and males of reproductive potential Lactation Unknown if distributed in human breast milk Because of the potential for serious adverse reactions in a breastfed infant, advise women not to breastfeed during treatment and for 2 weeks after the final dose

Interactions

Increased AUC w/ strong/moderate CYP3A inhibitors & P-glycoprotein (P-gp) inhibitors. Reduced exposure w/ strong CYP3A inducers.

Adverse Effects

Side effects of Cobimetinib : >10% Increased creatinine (99.6%) Increased CPK (79%) Increased AST (73%) Lymphopenia (73%) Increased alkaline phosphatase (71%) Anemia (69%) Increased ALT (68%) Hypophosphatemia (68%) Increased GGT (65%) Diarrhea (60%) Photosensitivity (46%) Hypoalbuminemia (42%) Nausea (41%) Hyponatremia (38%) Pyrexia (28%) Hyperkalemia (26%) Hypokalemia (25%) Hypocalcemia (24%) Vomiting (24%) Thrombocytopenia (18%) Acneiform dermatitis (16%) Hypertension (15%) Vision impaired (15%) Alopecia (15%) Stomatitis (14%) Hemorrhage (13%) Chorioretinopathy (13%) Retinal detachment (12%) Hyperkeratosis (11%) 1-10% Chills (10%) Erythema (10%)

Mechanism of Action

Reversible inhibitor of mitogen-activated protein kinase (MAPK)/extracellular signal regulated kinase 1 (MEK1) and MEK2 MEK proteins are upstream regulators of the extracellular signal-related kinase (ERK) pathway, which promotes cellular proliferation BRAF V600E and K mutations result in constitutive activation of the BRAF pathway, which includes MEK1 and MEK2 Cobimetinib and vemurafenib target 2 different kinases in the RAS/RAF/MEK/ERK pathway; compared with either drug alone, coadministration resulted in increased apoptosis in vitro and reduced tumor growth in mouse implantation models of tumor cell lines harboring BRAF V600E mutations Cobimetinib also prevented vemurafenib-mediated growth enhancement of a wild-type BRAF tumor cell line in an in vivo mouse implantation model