Luspatercept
Indications
Luspatercept is used for:
Anemia Related Beta-Thalassemia
Adult Dose
Anemia Related Beta-Thalassemia
Erythroid maturation agent indicated for anemia in adults with beta thalassemia who require regular red blood cell (RBC) transfusions
1 mg/kg once SC q3weeks initially
Increase to 1.25 mg/kg maximum if patient does not achieve reduction in RBC transfusion burden after at least 2 consecutive doses (6 weeks) at starting dose of 1 mg/kg
If predose hemoglobin (Hgb) ?11.5 g/dL and Hgb level not influenced by recent transfusion, delay dosing until Hgb is ?11 g/dL
If patient loses response to drug after initial response, search for causative factors (eg, bleeding event); if typical causes for lack of loss of response excluded, follow dosing recommendations for management of patients with insufficient response to therapy
Discontinue therapy if no decrease in transfusion burden after 9 weeks of treatment (3 doses) at maximum dose level or if unacceptable toxicity occurs at any time
Hepatic impairment
Mild-to-severe (total bilirubin ?ULN and AST or ALT >ULN, or total bilirubin >ULN and any AST or ALT): No clinically significant differences observed in pharmacokinetics of luspatercept
AST or ALT>3x ULN: Pharmacokinetics are unknown
Child Dose
Renal Dose
Renal impairment
Mild-to-moderate (eGFR 30-89 mL/min/1.73m2): No clinically significant differences observed in pharmacokinetics of luspatercept
Severe (eGFR <30 mL/min/1.73m2): Pharmacokinetics are unknown
Administration
Reconstitution
Reconstitute with sterile water for injection only
25-mg vial: Add 0.68 mL of sterile water for final concentration of 50 mg/mL for deliverable volume of 0.5 mL
75-mg vial: Add 1.6 mL of sterile water for final concentration of 50 mg/mL for deliverable volume of 1.5 mL
Allow reconstitution to stand for 1 min
Discard needle and syringe used for reconstitution; do not use for SC administration
Gently swirl vial in circular motion for 30 sec; stop swirling and let vial sit in upright position for 30 sec
Inspect for undissolved particles in solution; if undissolved powder is observed, repeat step above until powder completely dissolved
Invert vial and gently swirl in an inverted position for 30 seconds; bring vial back to upright position, and let it sit for 30 sec; repeat 7 more times to ensure complete reconstitution of the vial
Visually inspect for particulate matter and discoloration prior to administration whenever possible; drug is a colorless to slightly yellow, clear to slightly opalescent solution that is free of foreign particulate matter; discard if foreign particulate matter observed
SC Administration
Calculate exact total dosing volume of 50 mg/mL solution required for patient
If reconstituted vial was refrigerated, remove from refrigeration 15-30 min prior to injection to allow solution to reach room temperature for a more comfortable injection
Slowly withdraw dosing volume of reconstituted solution from single-dose vial(s) into a syringe
Divide doses requiring larger reconstituted volumes (ie, >1.2 mL) into separate similar volume injections and inject into separate sites; if multiple injections required, use a new syringe and needle for each SC injection
Administer injection subcutaneously into upper arm, thigh, and/or abdomen
Contra Indications
Precautions
Increased risk of thrombosis/thromboembolism in patients with beta thalassemia; patients with known risk factors for thromboembolism, eg, splenectomy or concomitant use of hormone replacement therapy, may be at further increased risk; consider thromboprophylaxis in patients with beta thalassemia at increased risk;.monitor for signs and symptoms of and institute treatment promptly
Monitor for hypertension; monitor blood pressure prior to each administration; manage new-onset hypertension or exacerbations of preexisting hypertension using anti-hypertensive agents
May cause fetal harm; advise females of reproductive potential of potential risk to fetus and use of effective contraception
Pregnancy-Lactation
Pregnancy
There are no available data on drug use in pregnant women to inform a drug-associated risk of major birth defects, miscarriage, or adverse maternal or fetal outcomes
Pregnancy testing recommended for females of reproductive potential before starting treatment
Animal data
Based on findings in animal reproduction studies, therapy may cause fetal harm when administered to a pregnant woman; in animal reproduction studies, administration to pregnant rats and rabbits during the period of organogenesis resulted in adverse developmental outcomes, including embryofetal mortality, alterations to growth, and structural abnormalities at exposures 13 times (rats) and 18 times (rabbits) of maximum recommended human dose (MRHD); advise pregnant women of potential risk to a fetus
Contraception
Females of reproductive potential: Use effective contraception during treatment and for at least 3 months after last dose; may cause embryofetal harm when administered to pregnant women
Infertility
Females: Based on findings in animals, therapy may impair female fertility; adverse effects on fertility in female rats were reversible after a 14-week recovery period
Lactation
Luspatercept-aamt was detected in milk of lactating rats
When a drug is present in animal milk, it is likely the drug will be present in human milk
There are no data on the presence of luspatercept in human milk, the effects on the breastfed child, or the effects on milk production
Owing to the potential for serious adverse reactions in the breastfed child, advise that breastfeeding is not recommended during treatment and for 3 months after the last dose
Interactions
Adverse Effects
Side effects of Luspatercept :
>10%
All grades
Total bilirubin ≥2x ULN (64%)
Headache (26%)
Arthralgia (19%)
Fatigue (14%)
Abdominal pain (14%)
Cough (14%)
Diarrhea (12%)
ALT >3x ULN (12%)
Dizziness (11%)
AST >3x ULN (11%)
1-10%
All grades
Nausea (9%)
Influenza (9%)
Alkaline phosphatase ≥2x ULN (8%)
Hypertension (8%)
Hyperuricemia (7%)
Direct bilirubin >2x ULN (6%)
Viral upper respiratory tract infection (6%)
Grade >3
Hyperuricemia (3%)
Hypertension (2%)
<1%
Grade ≥3
Diarrhea (<1%)
Headache (<1%)
Viral upper respiratory tract infection (0.4%)
Mechanism of Action
Drug is a recombinant fusion protein that diminishes Smad2/3 signaling by binding several endogenous TGF-beta superfamily ligands
In a model of beta thalassemia, drug decreased abnormally elevated Smad2/3 signaling and improved hematology parameters associated with ineffective erythropoiesis in mice
Treatment promoted erythroid maturation through differentiation of late-stage erythroid precursors (normoblasts) in mice