Trifluridine + Tipiracil
Indications
Trifluridine + Tipiracil is used for:
Indicated for treatment of patients with, metastatic colorectal cancer, Gastric Cancer
Adult Dose
Oral
Colorectal Cancer
Indicated for treatment of patients with metastatic colorectal cancer who have been previously treated with fluoropyrimidine-, oxaliplatin- and irinotecan-based chemotherapy, an anti-VEGF biological therapy, and an anti-EGFR therapy (if RAS wild-type)
Dose is based on trifluridine component rounded to the nearest 5-mg increment
35 mg/m² PO BID on Days 1-5 and Days 8-12 of each 28-day cycle; not to exceed 80 mg/dose
Continue until disease progression or unacceptable toxicity
Gastric Cancer
Indicated for treatment of patients with metastatic gastric or gastroesophageal junction adenocarcinoma previously treated with ?2 prior lines of chemotherapy that included a fluoropyrimidine, a platinum, either a taxane or irinotecan, and if appropriate, HER2/neu-targeted therapy
Dose is based on trifluridine component rounded to the nearest 5-mg increment
35 mg/m² PO BID on Days 1-5 and Days 8-12 of each 28-day cycle; not to exceed 80 mg/dose
Continue until disease progression or unacceptable toxicity
Hepatic impairment
Mild (total bilirubin [TB] ULN, or TB <1-1.5x ULN and any AST): No dosage adjustment required
Moderate-to-severe: Not studied
Child Dose
Renal Dose
Renal impairment
Mild-to-moderate (CrCl 30-89 mL/min): No dosage adjustment required; however, patients with moderate impairment (30-59 mL/min) may require dose modification for increased toxicity
Severe (CrCl <30 mL/min): Not studied
Administration
Oral Administration
Dose is based on trifluridine component rounded to nearest 5-mg increment
Administer with food; swallow whole
Do not take additional doses to make up for missed or held doses
Contra Indications
Precautions
Severe and life-threatening myelosuppression reported, including anemia, neutropenia, thrombocytopenia, and febrile neutropenia; obtain CBC count prior to and on Day 15 of each cycle, or more frequently as indicated
Based on animal studies and its mechanism of action, trifluridine/tipiracil can cause fetal harm when administered to pregnant women
Pregnancy-Lactation
Pregnancy
Based on animal data and its mechanism of action, drug can cause fetal harm
Embryo-fetal lethality and embryo-fetal toxicity occurred in pregnant rats when given during gestation at doses resulting in exposures lower than or similar to human exposures at the recommended clinical dose
There are no available data use in pregnant women
Verify pregnancy status in females of reproductive potential before initiating treatment
Contraception
Females of reproductive potential: Use effective contraception during treatment and for >6 months after final dose
Males with female partners of reproductive potential: Use condoms during treatment and for >3 months after final dose
Lactation
There are no data on the presence of trifluridine, tipiracil or its metabolites in human milk or its effects on the breastfed child or on milk production
In nursing rats, trifluridine and tipiracil or their metabolites were present in breast milk
Because of the potential for serious adverse reactions in breastfed children, advise women not to breastfeed during treatment and for 1 day following the final dose
Interactions
Adverse Effects
Side effects of Trifluridine + Tipiracil :
>10% (Metastatic Colorectal Cancer)
Anemia (77%)
Neutropenia (67%)
Asthenia/fatigue (52%)
Nausea (48%)
Thrombocytopenia (42%)
Decreased appetite (39%)
Neutropenia, Grade 3 or 4 (38%)
Diarrhea (32%)
Vomiting (28%)
Infections (27%)
Pyrexia (19%)
Anemia, Grade 3 or 4 (18%)
>10% (Metastatic Gastric Cancer)
Neutropenia (66%)
Anemia (63%)
Neutropenia, Grade 3 or 4 (38%)
Nausea (37%)
Decreased appetite (34%)
Thrombocytopenia (34%)
Vomiting (25%)
Diarrhea (23%)
Infections (23%)
Anemia, Grade 3 or 4 (19%)
Mechanism of Action
Trifluridine: Thymidine-based nucleoside analog that incorporates into DNA, interferes with DNA synthesis, and inhibits cell proliferation
Tipiracil: Thymidine phosphorylase inhibitor; inclusion of tipiracil increases trifluridine exposure by inhibiting its metabolism by thymidine phosphorylase